What may result from inadequate management of phosphorus levels in renal patients?

Inadequate management of phosphorus levels in renal patients can lead to potential vascular calcification. When phosphorus levels are not appropriately controlled, particularly in individuals with chronic kidney disease, the balance of calcium and phosphorus in the body can be disrupted.

Elevated phosphorus levels can cause the calcification of blood vessels and other tissues, a condition often referred to as vascular calcification. This occurs because the kidneys are unable to excrete excess phosphorus effectively, leading to its accumulation in the blood. High phosphorus levels can suppress the synthesis of active vitamin D, which in turn reduces the absorption of calcium from the diet. The result can be an imbalance that promotes calcification in vascular tissues, which increases the risk of cardiovascular complications—a significant concern for renal patients.

Other choices, such as improved bone density, increased muscle mass, or enhanced kidney function, do not accurately reflect the consequences of poor phosphorus management. In fact, high phosphorus levels typically contribute to secondary hyperparathyroidism, which can weaken bone density over time, rather than improve it.